Epidemiology, clinical manifestations, diagnosis...

Klebsiella pneumonia

is a member of the genus Klebsiella Enterobacteriaceae and belongs to the normal flora of human mouth and intestines. Infections, K. pneumonia, usually nosocomial and occur mainly in patients with low resistance. An important exception to these general observations outpatient primary invasive liver abscess syndrome that can occur in patients without major diseases and attract primarily been described in East Asia, including Taiwan [

]. (See below

).

Epidemiology, clinical manifestations, diagnosis and treatment of infections caused by K. pneumonia will be considered here. Microbiology and pathogenesis of infection of C. pneumonia are discussed separately. (See)

People are the main reservoir for Klebsiella pneumonia, although the organisms can also be found in soil and water. K. pneumonia in water coast may be zoonotic pathogens of marine mammals [

]. Carrier rate of K. pneumonia in humans ranges from 5 to 38 percent in stool samples and from 1 to 6 percent strattera no prescritpion in the nasopharynx, Klebsiella species is rare in the skin [

]. Higher rates of nasopharyngeal carriage occurred in outpatient alcoholism [

]. Carrier rates significantly increased in hospitalized patients who reported rates of 77 percent in the feces, 19 percent in the throat, and 42 percent on hand [

]. Higher rates of colonization primarily associated with the use of antibiotics [

]. This increased prevalence of clinically important, since in one report, Klebsiella nosocomial infection is four times higher chair carriers compared with noncarriers [

]. .

Plasmids coding for resistance to antibiotics...

Some genes of resistance to antimicrobial but not found is

on the bacterial chromosome. Others were found to lie on plasmids. Plasmids coding for resistance to antibiotics is often called resistance

factors, R-factor or R-plasmids. R-plasmids can encode >> << resistance to multiple unrelated antibiotics. Some R-plasmid independently transmitted and can move from strain to strain

, even among different bacterial genera. Other R-plasmids,

, while not itself being transferred can be mobilized other plasmids,

not necessarily encode resistance to antibiotics. In addition,

antibiotic purchase strattera resistance genes are often located in transposons. They can move more or less at random bacterial genome. Genes encoded by transposons so can spread very easily, because

many mobile elements can be associated with contagious >> plasmids. << Thus, genes of resistance to antibiotics can be easily

common. Control of antimicrobial resistance in pathogenic microbes

is one of the biggest challenges currently facing the medical

Microbiology. If we fail, we will surely enter the following antibiotics

era. . << >>

Without treatment, toxins from bacteria ...

Carnivorous bacteria leads to death within 30 to 40 percent of cases [Source:]. People strattera 10mg have experienced this situation, but battles fought and paid through the emotional and physical scars. The only treatment for carnivorous bacteria of high doses of antibiotics immediately as to prevent the spread of infection. Bacteria are so fast that any damaged or dying flesh must be removed to prevent the spread of bacteria. Without treatment, the toxins from the bacteria can spread throughout the body, causing toxic shock, which means that the body begins to shut down, lower blood pressure and eventually cause death. Some patients with flesh bacteria reached the stage of loss of consciousness and the need for life-support measures. Tissue damage may need surgery - skin grafts or even amputation. .